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Translatomics for eiF2A, rice, and synonymous mutations

Recent Publications Harnessing the Power of Translatomics Every week we provide a digest of a small number of recent interesting papers in the field of translatomics. In this week’s Sunday papers, Roiuk et al. use ribosome profiling to define the functional contribution of human eIF2A in translation initiation and uORF‑mediated control, using HeLa cells. Xi […]

Translatomics for m6A, m3C and ncRNAs

Recent Publications Harnessing the Power of Translatomics Every week we provide a digest of a small number of recent interesting papers in the field of translatomics. In this week’s Sunday papers, Linder et al. find that mcm5s2U tRNA modification counteracts m6A-dependent mRNA decay. Gao et al. develop a novel single nucleotide resolution method m3C-IP-seq for […]

Translatomics for RACK1, FAK and NAT10

Recent Publications Harnessing the Power of Translatomics Every week we provide a digest of a small number of recent interesting papers in the field of translatomics. In this week’s Sunday papers, Challa et al. highlight that MARylation of ribosomal protein RACK1 functions as a key regulator of stress granule formation. Kellett et al. study that Focal Adhesion Kinase (FAK) […]

Translatomics for proline, eIF6 and neoantigens

Recent Publications Harnessing the Power of Translatomics Every week we provide a digest of a small number of recent interesting papers in the field of translatomics. In this week’s Sunday papers, Reepmeyer et al. study that substituting Api137’s prolines with 4-fluoro-l-proline altered ribosome binding and activity. Roshan et al. investigate that targeting eIF6 sequesters inactive 80S ribosomes by disrupting […]

Translatomics for micropeptides and antigens

Recent Publications Harnessing the Power of Translatomics Every week we provide a digest of a small number of recent interesting papers in the field of translatomics. In this week’s Sunday papers, Xu et al. identify that a LINC00472-derived micropeptide suppresses non‑small cell lung cancer (NSCLC) proliferation and migration by disrupting HDAC2/SP1 interaction. Nah et al. show that microprotein SMIM26 […]